Many times you may have acne outbreaks and worry about it because you should assist a social gathering, school presentation or go on a romantic date. What to do about the acne outbreaks if you have just a couple of days in your hands?

It is certainly troublesome to know that you will have to assist some important event with pimples on your face. But in search of quick cures, you may waste precious money. There are no rapid miracle cures for acne, but there are a few remedies that help to clear acne soon. Let's find out.

If you have a large cyst that is filled with pus, the best method would be to get it injected with steroids. Your doctor will do that. Steroids dry out acne in few days.

If you have spots all over the face, you can use a combination of benzoyl peroxide and AHAs. Benzoyl peroxide can heal the acne and AHAs will peel away the devitalized skin layers to minimize the redness. You could also use products containing salicylic acid. It is a beta hydroxy acid and will treat the acne pimples fast.

If you have pus-filled spots and believe that those spots could get infected, you can ask your doctor for oral antibiotics. Antibiotics applied orally will eliminate all the propionibacterium acnes bacteria that are infecting your acne cysts.

Typical Concerns On Acne & Acne Treatment

Exaggerated sebum production: At adolescence, increasing levels of androgens, the main sebotrophic hormone, start to drive an increase in sebum production. However, while androgenic stimulation is crucial in the pathogenesis of acne, the typical acne patient does not have significant endocrine abnormalities. Hormonal therapy is not indicated in the initial management of mild to moderate acne, although females who require oral contraception can be candidates for anti-androgen therapy early in the course of treatment.

Unusual desquamation of the follicular epithelium: In acne, keratinocytes hyperproliferate and accumulate within the sebaceous follicle. As these aberrantly peeled cells accumulate in the sebaceous follicle, they lead to microcomedo formation. The microcomedo, is the initiator to all acne lesions and is present in 80% of acne papules but is imperceptible to the unaided eye. However, as the already blocked follicle starts to fill with lipids, bacteria and cell detritus, the microcomedo evolves to open or closed comedones (blackheads and whitehead, respectively), both of which are non-inflammatory lesions. If P. acnes grows, inflammatory mediators are generated and inflammatory papules and pustules appear.

Bacterial growth: The microenvironment of the follicle in acne is prone to population with P. acnes. This leads to inflammation and the formation of the evident papules and pustules with which acne patients commonly present to dermatologists.

Inflammation: Inflammation in acne occurs as an outcome of hormonal and cellular defensive reactions to P. acnes growth. It has been suggested that changes in sebum production or structure irritate infundibular keratinocytes leading to the liberation of interleukin 1a (IL-1a). Opening of the follicular tube leads to the extravasation of lipids, corneocytes and bacteria into the skin, causing more inflammation.